2016-05-30 01:20
epristeride翻译
爱普列特
音标读音
英式:[ɪprɪstə'raɪd]美式:[ɪprɪstə'raɪd]
基本意思
[医]依立雄胺<睾酮还原酶抑制药>
形近的词
变形
例句
- Objective To study the uroflometry of benign prostatic hyperplasia after the treatment of epristeride.
目的：研究服用爱普列特治疗良性前列腺增生症的尿流指标的改变。
- Aim To improve the synthesis of epristeride, a potent human 5a-reductase inhibitor.
目的改善5a-还原酶抑制剂爱普列特的合成方法。
- Objective To find out the changing in serum concentration of sex hormones after the treatment of epristeride and finasteride and to provide reference for medicine treatment of BPH.
目的通过爱普列特和保列治治疗后患者血清性激素水平变化的研究，为临床提供用药参考。
- Clinical Efficacy of Epristeride Plus Hytrin in the Treatment of Benign Prostatic Hyperplasia
爱普列特与高特灵联合治疗良性前列腺增生症临床疗效观察
- CONCLUSIONS: The pharmacokinetic profile of epristeride in old man was same as in young man with same dose level.
结论：爱普列特在老年人体内的药代动力学特性在同剂量下与青年人的一致。
- Effect of epristeride for the treatment of benign prostatic hyperplasia
爱普利特治疗前列腺增生患者的疗效观察
- CONCLUSION: Epristeride may up-regulate the expression of T β R ⅱ to induce apoptosis of prostatic epithelial cells.
结论：爱普列特诱导前列腺上皮细胞凋亡可能与上调TβRⅡ表达有关。
- [ CONCLUSION] High doses of Epristeride and Finasteride suppressed the development of prostate and reduce weight of tetis.
[结论]较大剂量非那甾胺与爱普列特可以抑制雄性大鼠前列腺生长、减少睾丸重量；
- Pharmacology and Toxicology Study of Epristeride: A Novel Drug against Benign Prostate Hyperplasia
新型抗前列腺增生药爱普列特（Epristeride）药理和毒理研究
- Apoptosis induce effect of epristeride on benign prostatic hyperplasia model cells in vitro
爱普列特对良性前列腺增生体外细胞模型的凋亡诱导作用
- Objective Study the efficacy and safety of epristeride, a new uncompetitive 5 α-reductase inhibitor, in the treatment of benign prostatic hyperplasia ( BPH).
目的探讨新型非竞争性5α还原酶抑制剂爱普列特（epristeride）治疗良性前列腺增生（BPH）的有效性及安全性。
- He grew very animated, quite the amiable St. Preux at fifty. Clinical Efficacy of Epristeride Plus Hytrin in the Treatment of Benign Prostatic Hyperplasia
他变得生气勃勃起来，活象五十岁的圣·普瑞那么可爱。爱普列特与高特灵联合治疗良性前列腺增生症临床疗效观察
- AIM: To establish a benign prostatic hyperplasia ( BPH) cells model in vitro and investigate the mechanism of epristeride to BPH cells based on this model. METHODS: Establishing the primary cultural process of rat prostatic cells.
目的：拟建良性前列腺增生体外细胞模型，并探讨爱普列特（epristeride）对前列腺增生模型中前列腺细胞作用机制。
- METHODS: RT-PCR and Western blot were used to quantitatively detect the mRNA and protein expressions of T β R ⅱ in rat prostatic epithelial cells treated or untreated with epristeride. Immunocytochemical staining method was used to qualitatively analyze the expression of T β R ⅱ protein.
方法：逆转录PCR法及westernblot定量检测体外培养前列腺上皮细胞TβRⅡmRNA及蛋白表达，免疫组织化学染色定性分析爱普列特对前列腺上皮细胞TβRⅡ表达的影响。
- AIM: To study molecular mechanism of epristeride in the treatment of benign prostatic hyperplasia and discuss the possibility of using prostate acid phosphatase ( AGP) as a marker of the atrophy of prostatic gland in vivo.
目的：研究爱普列特是否通过诱导前列腺细胞凋亡来治疗前列腺良性增生。探讨以前列腺酸性磷酸酶作为前列腺萎缩标志的可行性。
- Conclusion: High dosages of the 5 α-reductase inhibitor, Epristeride, can suppress the development of the prostate and reduce the weight of the testis, decrease dihydrotestosterone, and inhibit spermatogenesis and productive function in male rats.
结论：大剂量高选择性5α还原酶抑制剂（爱普列特）可抑制雄性SD大鼠前列腺、睾丸重量，抑制精子生成并降低雄性大鼠生育力。
- Inhibitory effect of epristeride on the expression of prostate specific antigen and Bcl-2 protein in human prostatic carcinoma LNCaP cells in vitro
依立雄胺对前列腺癌细胞（LNCaP）前列腺特异性抗原和Bcl-2蛋白体外表达的抑制作用
- A Multicentre Clinical Trial on Epristeride for Treatment of Benign Prostate Hyperplasia
爱普列特治疗良性前列腺增生Ⅱ期临床研究
- Methyl 3 β-hydroxy-5-androstene-17 β-carboxylate, the key intermediate of finasteride and epristeride, was prepared from pregnenolone acetate by bromoform reaction and esterification with 69% overall yield.
以孕烯醇酮醋酸酯为起始原料，经溴仿反应、酯化反应制得3β-羟基-5雄甾烯-17β-羧酸甲酯，总收率69%。
- Objective: To identify the role of 5 α-reductase in the spermatogenesis of male rats by studying the effect of two 5 α-reductase inhibitors, Epristeride and Finasteride, on the spermatogenesis in male Sprague-Dawley ( SD) rats.
目的：研究高选择性5α还原酶抑制剂（爱普列特）在较大剂量时对雄性SD大鼠生精功能的影响。
- CONCLUSION: The molecular mechanisms of prostatic epithelial cell atrophy induced by epristeride might be associated with alteration in the expression of growth factor receptors such as EGF and IGF-ⅰ.
结论：爱普列特抗前列腺增生的分子机制与纠正增生时异常表达的生长因子有关。
百科解释
网络意思